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http://www.ebi.ac.uk/Tools/services/web/toolresult.ebi?jobId=clustalo-I20170607-171424-0715-19580227-es&analysis=phylotree

Explain the evalution relationship between those species and what dose 0 number in some species means?

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Jarrod Robel
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10 Jan 2018
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Related questions

Creat statistical test help please

Conduct an appropriate statistical test to see if there significant difference in the number of nutrition points collected for the DEAD (starved and killed) prey between high and low resource abundances. (Hint: you are testing for a significant difference between two population means.) Provide the complete outputs and highlight the appropriate P-value based on your hypotheses. Remember to evaluate your P-value and write out the conclusion.

---> What are some of the ecological reasons that may explain this result? Interpret your results in the context of optimal foraging theory and whether the prey is willing to take risks. (5 pts)

CLASS DATA OBTAINED BELOW:

 Mean nutrition points gathers vs. outcomes (alive & dead)
alive high alive low dead high dead low
12 4 7
12 4 7
13 7 0
6 7
2 4
2
4
3
MEAN alive high MEAN alive low MEAN dead high MEAN dead low
37 0 23 34

Read the following passage focusing your attention on the scientific procedures and scientific abilities illustrated in it. Think about what types of experiments (observation, testing or application) are described. Identify the hypothesis, predictions, outcomes of experiments and assumptions that appear in the passage. The standard explanation of how humans originally acquired AIDS is that HIV (human immunodeficiency virus) originally was transmitted to humans from monkeys or apes carrying SIV, simian immunodeficiency virus. How could the transmission have occurred? Several similar hypotheses involve an African hunter coming in contact with monkey blood. For example this could have occurred when a hunter butchering a monkey, got some of its blood in a cut, or when a human ate undercooked monkey meat, or when a monkey bit a human. Depending on one's assessment of the genetic diversity of HIV over time, this transmission might have occurred only a few decades ago or, alternatively, hundreds or thousands of years ago. If the latter is correct then AIDS could have been restricted to remote African villages until recent decades when trade and international travel led to the current pandemic. There are however other alternative explanations for the spread of AIDS. One of them is that AIDS arose from contaminated polio vaccines used in Africa from 1957-59. Why is this a plausible explanation? Polio vaccines are cultured on apes’ kidneys, so contamination of a batch of vaccine by an SIV is a possibility. There is a precedent: in the 1950s and early 1960s millions of doses of polio vaccine were contaminated by another simian virus, SV-40. The vaccine alleged to be the origin of AIDS was given to hundreds of thousands of people in central and west Africa from 1957-1959. These locations later had some of the world's highest levels of HIV infection. The timing is also right, since the earliest known cases of AIDS date from the 1960s; the earliest HIV-positive blood sample was collected in Kinshasa in 1959. At the time, there was no way of testing for the presence of SIVs, which were only discovered in the 1980s. Another factor adds plausibility to the theory: the vaccine was given to many infants less than a month old, in an extra high dose. Since the immune systems of infants are not fully developed, this is an excellent way to help a virus jump the species barrier. Some scientists argue that the above explanation is not necessarily correct but that it is worth taking seriously. There are a number of ways to test it. One is to analyze stocks of polio vaccine; if the explanation is correct, then it is probable that some of the vaccines will carry SIV. Another is to scrutinize records of early polio vaccination campaigns in Africa. If it is true that the immunodeficiency virus passed to humans by way of those vaccines, the areas where the campaigns took place would be the same as those where the initial sprouts of the disease occurred and where the larger number of cases occurred during the early stages of the pandemic. Another is to analyze blood samples in Africa from the 1950s and earlier (pre polio vaccination): if the immunodeficiency virus was transmitted via the polio vaccine, HIV should not be found. Adapted from “Political refutation of a scientific theory: the case of polio vaccines and the origin of AIDS”. Brian Martin, published in Health Care Analysis, Vol. 6, 1998, pp. 175-179. Now reread the passage and answer the following questions.

5. For each experiment, write an outcome that would disprove the hypothesis of AIDS passing from monkeys to humans through polio vaccines.

6. What outcomes of the experiments would fail to disprove the hypothesis of AIDS passing from monkeys to humans through polio vaccines?explain.

7.What outcomes would prove the hypothesis? Hint: This is not the same as question 6. Think about what would you need to do to 100% prove a hypothesis.

8. For what reasons do scientists need to conduct testing experiments?

In a small paragraph, compare and summarize these two articles.

Article 1 Description

This article is an observational study, which discusses how frequency dependent selection affects two phenotypes of a predator through differential guarding response. The researcher examines the ratio of handedness of two samples of P. microlepis collected from two adjacent sites along Lake Tanganyika’s northwestern shoreline for a period of eleven years. The process of observations includes observing the correspondence between the handedness of P. microlepis and their targeted prey (Hori, 1993). The researcher carried observations to identify the direction from which P. microlepis attacked the flank of their prey. Moreover, the researcher used a gill net to catch each P. microlepis that attacked the lure. Catching the P. microbes made it possible for the researcher to observe its features and determine its handedness. The researcher determined the handedness of the scales of P. microlepis by observing the fish under a microscope.

During the period of study, Hori (1993) established that the ratio of handedness of P. microbes remained at 0.5 for each one-to-two year intervals throughout the eleven years of the study. Further investigations on temporal changes in handedness ratio showed that the ratio oscillated with amplitude of 0.15 within a five-year period (Hori, 1993). These observations were made for the two species of P. microlepis under investigation. The researcher concludes that the direction of mouth opening of scale eating cichlid fish in Lake Tanganyika is determined by the genetics of the fish. The researcher demonstrates that frequency-dependent natural selection determines the number of left-handedness or right-handedness fish. Based on researcher’s observations, when right-handed fish attack from behind, they snatch the scales from their prey from the left flank (Hori, 1993). On the other hand, when left-handed fish attack their prey from behind, they attack the right flank (Hori, 1993). Therefore, fish with rare phenotypes are more successful as predators than ones that have the more predominant phenotype. This study can be used to guide researchers who are interested in seeking ways of increasing the population of different species of fish. It can also be used as a basis for investigations on how fish type determines its survival tactics or predator tactics.

Article 2 Description

The study was an experimental analysis which sought to quantify phosphoglucose isomerase (Pgi) frequency change over a period of time and compare Pgi genotypes based on physiological responses to high temperatures and subzero thermal extremes encountered by beetles in nature. The research seeks to establish how the local climate in Bishop relates to changes in allele frequency at Pgi and other allozyme loci. It also seeks to establish if ambient temperature of beetle habitants rise to levels that expose beetles to stressful temperature. The researcher also seeks to establish how Pgi genotypes differ physiologically when exposed to subzero temperatures. By answering these questions, the researcher will establish the relationship between changes in enzyme genotype frequency and regional climate changes in addition to investigating major physiological mechanisms through which animals respond to change in climate.

To answer research questions and test research hypothesis, the researchers carried out several experiments and analyses of data. The experimental study involved collecting adult beetles that had emergent recently from pupae for a period of one year between August 1988 and August 1989 (Rank & Dahlhoff, 2002). Their genotype was determined at five different polymorphic enzyme loci. Some of the things analyzed during the study include fluctuations in climate and allele frequency change; daily and annual mean values of air temperature, Effects of Pgi genotype on HSP70 expression, and quantification of Pgi genotype on HSP70 expression among other things (Rank & Dahlhoff, 2002). The study results revealed that although beetle populations were genetically different the FST-values for 1996 were greater for each enzyme locus than those for 1998 (Rank & Dahlhoff, 2002). Moreover, researchers established that the frequency of Pgi allele had increased by 11 percent, a trend that was not observed for all the other loci investigated. Therefore, the researchers conclude that Pgi genotype differences in thermal physiology account for Pgi allele frequency change in nature. Contrary to selected hypothesis that the frequency of Pgi allele should increase with decrease in temperature, the study established that the frequency of Pgi allele decrease with decrease in temperature. This work is important because it provides a basis for more research on how variations in temperature affect beetle populations. The study findings can be used to guide researchers who are interested in seeking ways of protecting endangered beetle populations in different climatic conditions.

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