🏷️ LIMITED TIME OFFER: GET 20% OFF GRADE+ YEARLY SUBSCRIPTION →
Log in
Biology
8
answers
1
watching
155
views

How will the cell shape alter if FtsZ is gradually depleted in an E. coli cell?

Watch
8
answers
1
watching
155
views

For unlimited access to Homework Help, a Homework+ subscription is required.

Unlock all answers

Get 1 free homework help answer.
Already have an account? Log in
Share
Already have an account? Log in
Share
Already have an account? Log in
Share
Already have an account? Log in
Share
Already have an account? Log in
Share
Already have an account? Log in
Share
Already have an account? Log in
Share
Already have an account? Log in
Share

Related textbook solutions

Molecular Cell Biology
8 Edition,
Lodish
ISBN: 9781464183393
Biology: Science for Life with Physiology
6 Edition,
Belk
ISBN: 9780134555430
Concepts of Biology
OER Edition,
Openstax
ISBN: 9781938168116
Biology
2nd Edition, 2013
Openstax
ISBN: 9781947172517
Essentials of Biology
5 Edition,
Mader
ISBN: 9781259660269

Related questions

What might be an evolutionary rationale forconservation of the ftsZ region(proteins involved in cell division)in various bacteria? [6 points]

I worked on other questions involving thecomparison of about 5 organisms with the ftsZ region having ahighly conserved region, however im not sure how to answer thisquestion. I was thinking function, but the whole "evolutionaryrationale" part is throwing me off from answering this questionstronger.

Any ideas? Please do not copy and pastewebsites with answers, id rather have someone who knows what theyare talking about so I can understand it. Thank you!

A. Since it takes 40 min. to replicate the E. coli chromosome,how can the cell have a cell cycle of 35 minutes?


B.What would happen to a bacterial chromosome during celldivision if it were not connected to the membrane?


C. How do eukaryotes divide their pairs of chromosomes into twoseparate cells?


D. How could you isolate a loss of function mutation is anessential gene when mutating the gene can kill the cell?


E. How is it possible that a single gene like ftsZ can havemutations that have different phenotypes?


F. What would minC mutant bacterial look like?


G.Why is recombination after replication only a problem fro abacterium that has a circular chromosome and not a linearchromosome?


H.Why do bacterial have to attach their chromosomes to themembrane for effective separation, whereas eukarotes do not?


I.Why is the classical signal transduction pathway so complex,why not simply have the growth factor receptor activate thetranscription factor?


J.What is the phenotype of a cell that is heterozygous for a RASoncogenic mutation?


K. How does p53 communicate with Rb?


L. What is the phenotype of a cell that is heterozygous for aloss of function p53 mutant?


M. Rb is only acted upon by Cyclin/CDK, how can it integrate DNAdamage signal from p53?

A. Since it takes 40 min. to replicate the E. coli chromosome,how can the cell have a cell cycle of 35 minutes?


B.What would happen to a bacterial chromosome during celldivision if it were not connected to the membrane?


C. How do eukaryotes divide their pairs of chromosomes into twoseparate cells?


D. How could you isolate a loss of function mutation is anessential gene when mutating the gene can kill the cell?


E. How is it possible that a single gene like ftsZ can havemutations that have different phenotypes?


F. What would minC mutant bacterial look like?


G.Why is recombination after replication only a problem fro abacterium that has a circular chromosome and not a linearchromosome?


H.Why do bacterial have to attach their chromosomes to themembrane for effective separation, whereas eukarotes do not?


I.Why is the classical signal transduction pathway so complex,why not simply have the growth factor receptor activate thetranscription factor?


J.What is the phenotype of a cell that is heterozygous for a RASoncogenic mutation?


K. How does p53 communicate with Rb?


L. What is the phenotype of a cell that is heterozygous for aloss of function p53 mutant?


M. Rb is only acted upon by Cyclin/CDK, how can it integrate DNAdamage signal from p53?

Weekly leaderboard

Home
Homework Help 3,900,000
Biology 460,000
Start filling in the gaps now
Log in