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So we are studying antibiotics and can you tell me how the following antibiotics work against bacteria and if the bacteria became resistant to the antibiotic, what is the most likely way it would do it?

Rocephin

Sumycin

Paromycin

Vancocin

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Bunny Greenfelder
Bunny GreenfelderLv2
28 Sep 2019
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Related questions

Watch the TEDEd video What causes antibiotic resistance ( https://ed.ted.com/lessons/how-antibiotics-become-resistant-over-time-kevin-wu )and answer the following questions: 1) How many more bacterial cells do you have in and on your body than human cells? 2) What are 2 beneficial things these bacteria do for us? 3) Name 2 ways that antibiotics work to kill bacteria or inhibit their growth. 4) State 2 alternative solutions scientists have created to combat antibiotic resistant bacterial infections.

My Cell Bio book is discussing how bacteria can become resistant to antibiotics. Antibiotics that are used to target important processes of protein synthesis in bacteria seem to be having problems that prevent them from being effective. One possible solution that was talked about in the book is using CRISPR to target these antibiotic resistant genes in bacteria.

So here is my question, how effective would this method be? My book mentions a few of the genes that may play a role in antibiotic resistance. Is it possible for CRISPR to target every gene mutation of every resistant bacteria? Would this reversal be quick enough to fight the growth of resistant colonies on a large scale?

Your doctor diagnosed your illness as a bacterial sinus infection and prescribed a course of antibiotics. You also gave informed consent to participate in the doctor’s research study on antibiotics. As part of the study you went to the clinic every day to have a sample from the infection taken and analyzed. Before starting on the antibiotics the bacteria were susceptible to the antibiotic; however later in the week after starting the antibiotic treatment the bacteria in your infection were resistant to the antibiotic. Results of DNA analysis of the bacteria causing your infection showed that the resistant bacteria and susceptible bacteria differed only by a gene that encodes the protein target of the antibiotic such that the antibiotic was unable to bind to the target site on the resistant bacterial cells..

Which of the following three hypotheses is most likely? Explain how the results support that hypothesis and what you learned about antibiotic resistance in week 4 topic “Antibiotic Resistance”. What is a possible mechanism for the change in the gene? What are at least three ways previously sensitive bacterial cells acquire the gene for gene for resistance (name and describe them)?

A) You developed a tolerance* for the antibiotic

B) The bacteria infecting you developed a tolerance* for the antibiotic.

C) The bacteria infecting you evolved to be resistant to the antibiotic.

*Tolerance is a nongenetic change in which the response to a specific drug and concentration is weaker after repeated use.

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