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  1. During an inflammatory response, the immune system may developmemory cells for the bacteria so that antibodies can aid theresponse in future infections. What cell is generating these memorycells?

    neutrophils

    Helper T cells

    memory or naive B lymphocytes

    macrophages

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Nelly Stracke
Nelly StrackeLv2
28 Sep 2019
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Related questions

1. Macrophages, dendritic cells, and neutrophils are all examples of __________

a. Phagocytes

b. Lymphocytes

c. Basophils

d. Parasites

2._______ is an antimicrobial peptide secreted by many types of animal cells. It breaks bonds in peptidoglycan and is most effective against _______.

a. Lysozyme i. Fungi

b. Histatin ii. Viruses

c. Lactoferrin iii. Gram positive bacteria

d. N-acetylglucosamine iv. Gram negative bacteria

3. Use of antibiotics can cause perturbations in this innate immune component.

a. Macrophages b. Dendritic Cells c. Toll-like Receptors d. Microbiota

4. Which potential outcome of complement activation seems to only effect bacteria of the genus Neisseria?

a. Initiation of inflammatory response b. Opsonization c. Membrane attack complex formation d. Apoptosis

11. Which type of immunityoccurs as a response to illness or vaccination?

Acquired active

Acquired passive

Natural


12. What are the uniquecharacteristics of the immune system?

repository, recognition, defense

self recognition, specificity, memory

interception, defense, notification

specificity, memory, defense


13. Which of the followingis not one of the three unique characteristics ofthe immune system?

memory

sensitivity

specificity

self-recognition


14. Which of the followingis an age related change associated with the immune system?

Increase in the number of macrophages and monocytes

The thymus continues to produce the same number of T cells withage

The rate of B-cell production declines with age

Immune memory declines with aging


15. A decrease in ________secretion causes a lowering of blood volume and blood pressure andan increase is body osmotic pressure.

FSH

GH

LDH

T3

ADH


16. Which of the followingstatements regarding antibodies is true?

they destroy antigens by engulfing them

IgA activates the complement system

IgE antibodies release histamine and cause inflammation

Antibodies are manufacture and by platelets


17. Which condition causesautoimmune damage to receptors for acetylcholine at theneuromuscular junction?

Multiple sclerosis

Rheumatoid arthritis

Myasthenia gravis

Crohn's disease


18. Which is true inregards to Immune memory?

the number of memory cells increase with age due to the fact thebody has been exposed to numerous antigens throughout life

the strength of the memory cells is not altered with aging butthe speed in which the memory cells responds decreases with age

the function of the memory cells is not really altered until thepatient reaches approximately eighty years of age

less decline in the ability to maintain memory that was producedduring youth or young adulthood, as oppose to memory that came onin older adulthood


19. All of the followingare ways to minimize the undesirable effects of aging on our immunesystem, except what?

diet low in complex carbs

regular vaccinations

seek early treatment when ill

wearing sun protection


20. Which type of immunesystem cell is formed from an unspecialized lymphocyte because ofthe influence of the thymus on the lymphocyte?

A-Cell

D-Cell

G-Cell

M-Cell

T-Cell

1. Why is the process of gene rearrangement used in creating antibodies and T-cell receptors (TCRs) so important?

A. This is how our systems produce memory of pervious exposures, a key trait in adaptive immune responses.

B. Without it, we would need a single gene for every antibody and TCR required to mount all the possible responses need ed during our lives

C. interactions with antigens stimulate cells to initiate these processes, building the best and strongest response possible to the antigen

D. this is the same DNA altering process that leads to cancer. Understanding it leads to better treatments for cancer

2. Which of the following is NOT an antimicrobial substance?

A. keratin

B. Lysozyme

C. Defensins

D. Lactoferrins

E. Peroxidases

3. Which of the following is NOT an outcome of fever?

A. Direct destruction of microbes

B. increased release of pro-inflammatory cytokines

C. increased production of lymphocytes

D. increased phagocytic killing by leukocytes

E. increased production of interferons

4. Which of the following pattern recognition receptors is/are anchored in the host cell membrane?

A. Only C-type lectin receptors (CLRs)

B. Only RIG-like receptors (RLRs)

C. TLR and CLRs

D. Only toll-like receptors (TLRs)

E. NLRs and RLRs

F. Only NOD-like receptors (NLRs)

5. Fever allows the body to fight microbial invaders by which of the following mechanisms?

A.Fever plays all of these roles in the body

B. Fever prevents microbes with lower optimum temperatures from growing, giving the immune system time to eliminate those cells

C. Fever increases the inflammatory response, but reduces the production of interferons unless the invader is a dsRNA virus

D. A moderate fever reduces the inflammatory response but stimulates phagocytic activity in neutrophils and eosinophils

E. Fever increases the multiplication of lymphocytes and enhances the release of substances that attract basophils that then engulf the microbial invader

6. According to the clonal selection theory

A. Each B cell is already programmed to produce a specific antibody

B. B cells producing autoantibodies are eliminated in the thymus

C. Self-reactive T cells are killed in the thymus AND B cells producingautoantibodies are eliminated in the thymus

D. Seld-reactive T cells are killed in the thymus

7. Apoptosis

A. is a form of programmed cell death AND is induced in target cells by effector T cytotoxic cells

B. refers to the transformation of B cells into plasma cells

C. is induced in target cells by effector T cytotoxic cell AND results specifically in T cell death

D. results specifically in T cell death AND refers to the transformation of B cells into plama cells

8.

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