Where do C3a and C3b come from
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1. C3a and C5a are proteins which effectively _____.
a.Opsonize invading pathogens
b.Recruit immune cells to infection site
c.Form pores in bacterial and fungal cell membranes
d.Cleave covalent bonds in peptidoglycan
2. Some pathogenic bacteria produce ______ which protect the organism against the oxidative burst from _________.
a.Catalase i. Phagocytic cells
b.Nitric oxide ii. Red blood cells
c.Superoxide Dismutase iii. Cytokines
d.C3 convertase iv. B Cells
3. What is extravasation?
a.The expansion of joints between endothelial cells allowing for the migration of immune cells and proteins access to infection sites.
b.Recruitment of macrophages to infected tissues.
c.The process by which Salmonella avoids killing by phagocytic cells.
d.Coating of a pathogen with C3b for uptake by macrophages.
4.Atypical hemolytic uremic syndrome results from mutations in which complement component?
a.Factor I
b.Factor H
c.C3b
d.CR1
Describe the following: endotoxin, exotoxin, C3a,Capsular proteins, Flagellar proteins (H antigen), Lipid A of theLPS in gram-negative cells and Peptidoglycan in gram-positivecells
What is extravasation?
a. The expansion of joints between endothelial cells allowing for the migration of immune cells and proteins access to infection sites.
b. Recruitment of macrophages to infected tissues.
c. The process by which Salmonella avoids killing by phagocytic cells.
d. Coating of a pathogen with C3b for uptake by macrophages.
Atypical hemolytic uremic syndrome results from mutations in which complement component?
a. Factor I
b Factor H
c. C3b
d. CR1
