KIN 321 Final: Final review
17 Aug 2016
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Document Summary
Fa either goes into the blood or it is re-esterified. Because of that the change in glycerol [ ] in blood is a marker of whole body lipolysis: lots of glycerol = a lot of lipolysis. What counters all this is insulin insulin is a storage hormone. Insulin has receptors on adipose tissue, binds, activates phosphodiesterase this breaks down cyclic amp. When you consume cho, that slightly bumps up your insulin slows mobilization. Consuming cho, increase cho use, increase rates of lipolysis inhibits. High rates of glycolysis = low ph. Sympathetic = sa node, av node, ventricular mitocardium: increases cardiac output by influencing hr and stroke volume, to affect stroke volume it has to affect mitocardium to increase contractility. Fat metabolism: hsl = responsible for mobilizing fats for lipolysis, cat1 = rate limiting, brings fat across outer mitochondrial membrane (cat2 brings across inner mitochondrial membrane)
