Physiology 1020 Study Guide - Final Guide: Basilar Membrane, Sinoatrial Node, Resting Potential

• School: Western University
• Department: Physiology
• Course: Physiology 1020
• Professor: John Bell

Document Summary

Higher concentrations of na+, ca++, and cl- on the outside. Higher concs of k+ on the inside. Gated channel protein: spot where atp can attach. All or nothing change intensity by increasing frequency. Back up to resting potential at -70mv. Saltatory conduction: ap moves faster (cid:523)jumps to nodes of ranvier(cid:524) because it"s. Activated gate closed at rest, inactivation gate open. Threshold reached, activation gate opens and na+ influx. Absolute refractory period: na+ channels are inactivated= no more aps. Relative refractory period: na+ channels resetting, cells is hyperpolarized= Voltage gates ca++ channels open= influx of ca++ Ca++ triggers fusions of ach filled vesicles into synaptic cleft. Ach binds to receptors located the muscle cells end plate. Ligand gated na+/k+ channels open= influx of na+ Epp depolarizes and voltage gated na+ channels open= ap. Ach broken down into choline and acetic acid by acetlycholinesterase. Whole muscle fascicle muscle cell myofibrils actin and myosin.