HUN-1201 Lecture Notes - Lecture 21: Ketogenesis, Glycogen, Acetyl-Coa

• School: FSU
• Department: Nutrition, Food and Exercise Sciences
• Course: HUN-1201
• Professor: Jennifer Farrell

Document Summary

All a(cid:373)i(cid:374)o acids have a(cid:374) (cid:862)r(cid:863) chai(cid:374) Can be stored as fat if there is an excess of proteins: ketogenic amino acids amino acids that can be turned into ketogenic intermediates which are then turned into ketones through ketogenesis (ketone formation) Carbohydrates glycogen (liver and muscle) fat (if glycogen stores are full) Fat stored as fat, carried to adipose tissue by lipoproteins. Proteins make new proteins, neurotransmitters, stored as fat if we have extra, least likely to be stored as fat. Insulin promotes storage, lowers blood glucose levels, glycogenesis (glucose to glycogen) Glucose goes to cells, anything not needed is stored as fat: fasting. Carbohydrates: glucagon hormone that is secreted during fasting, releases blood sugar into the bloodstream. Takes things out of storage, opposite of insulin essentially. Glycogen broken down into glucose, glycogenolysis, further broken down into acetyl coa (through glycolysis) which is then broken down into atp (through the krebs/tca cycle)