Kinesiology 2230A/B Lecture Notes - Lecture 2: Hypertrophic Cardiomyopathy, Blood Pressure, Catecholamine
16 Mar 2019
School
Department
Course
Professor
Document Summary
Left ventricular wall is thicker: most common cause of sudden death in youth, myofibril disarray that becomes more prevalent with age, chambers can"t eject blood, contractile protein signalling occurs - not getting enough co or sv, hypertrophy increases. Blood pressure and rhythmic exercise: diastolic pressure doesn"t change, directly associated with the elasticity of the arterial system. Cardiac structure and function: co affects o2 use, without any changes in co, o2 use increases, as hr goes down, ability to deliver o2 decreases. Factors affecting sv: contractibility - the force with which the heart contracts. Sv and edv: an increase of exercise intensity = increase in edv, as edv increases, sv increases. If we superimpose the effects of sympathetic stimulation on the initial edv response, we see that sv is greater at any edv. Increase in venomotor tone (vasoconstriction) increases venous return, which increases preload, and the smooth muscles of the veins will contract.
