Kinesiology 2230A/B Lecture Notes - Lecture 20: Atrioventricular Node, Sinoatrial Node, Stroke Volume

• School: Western University
• Department: Kinesiology
• Course: Kinesiology 2230A/B
• Professor: Dr. Belfry

Document Summary

This disease is too much heart tissue. Ventricular chamber is very small, effecting their stroke volumes. Most common cause of sudden death in young people (less than 30 years) Heart muscle cells originate and attach to adjacent fibres. Compress volumes of the heart as it pumps blood. Get myofibrilar disarrays when muscle fibres that are usually even lines become much more random. Fibres still contract but because of the disarray of fibres, get dysfunctional contractions. Hypertrophy would make it stronger and alleviate the problem. Electrical wave move through atria and arrives at av node. The av node is high in fat tissue and doesn"t conduct electrical current effectively so theres a delay. Full contraction of atria before contraction of ventricles. Move through bundle of his and perkinje fibres. The wave is reflecting electrical activity being initially at sa node. P wave is activity at atria- tissue being depolarized. Flat line is delay through av node.