AN SC311 Lecture Notes - Lecture 7: Glycogen Storage Disease, Lipoprotein Lipase, Very Low-Density Lipoprotein

• School: University of Alberta
• Department: Animal Science
• Course: AN SC311
• Professor: Benjamin Willing

Document Summary

I(cid:374) ru(cid:373)i(cid:374)a(cid:374)ts it"s the (cid:373)i(cid:272)ro(cid:271)es i(cid:374) the gut that (cid:272)a(cid:374) (cid:373)ake all the a. a that is (cid:374)eeded. The liver is no better than a non-ruminant. Basically this enzyme doesn"t work and they have build-up of glycogen. Allows for rapid release of glucose when needed. I(cid:374) a(cid:271)sorpti(cid:448)e state you do(cid:374)"t (cid:374)eed to (cid:373)ake glu(cid:272)ose out of a. a. Glucose can be taken up by all tissues and used as energy. They get stiff because of build-up of carbs in muscles. Do(cid:374)"t allo(cid:449) it (cid:271)e (cid:271)ra(cid:374)(cid:272)hed as (cid:373)u(cid:272)h as it should (cid:271)e. Vldl remnants go back to liver to be disposed of. When glucose is in the blood it is free and anything can take it up. But free fatty acids must be bound to something (hydrophobic) Has a specific receptor it needs to bind to. Phosphatase is not present in muscle therefore glucose cannot get out. Must be utilized 1st and changed to pyruvic and lactic acid.