NURS 2050 Lecture Notes - Lecture 18: Small-Cell Carcinoma, Vasoactive Intestinal Peptide, Vinca Alkaloid
19 Jan 2017
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Think of clinical examples of each of these concepts. ****please refer to the slides that illustrate the difference between apoptosis cell. A. key terms: excessive cellular proliferation, anaplasia, de-differentiation or un-differentiated, benign, malignant, direct invasion, seeding, metastases, proto-oncogenes, oncogenes, tumor suppressor genes (examples p53, rb etc), dna repair genes (e. g. parp). You must understand the functions of proto- oncogenes (the three ways they are activated), oncogenes, tumor suppressor genes and dna repair genes. Other terms are contact inhibition, encapsulated, carcinoma in situ, angiogenesis, paraneoplastic syndrome, and anorexia/cachexia. ****you must know the cell cycle and what is occurring in each of the phases, as well as the checkpoints. As seen in the slide presentation i posted on chemotherapy drugs, the mechanisms of these drugs classes is dependent on you understanding the cell cycle. Below are the questions we did from top. Hat in the class: charles has a gastrointestinal tumor.
