BIO 3347 Lecture 1: Renal Disease
Document Summary
Renal disease interferes with the normal capacity of nephrons to lter waster products of body metabolism. Short term renal disease requires basic nutritional support for healing rather than dietary restriction. Based on the nature of the disease process and individual responses. Clinical symptoms: massive edema, proteinuria (very large amount), distended abdomen, reduced plasma protein level, body tissue wasting. Protein intake to meet nutritional/growth needs (without excess) About 75% of nephrotic syndrome cases result from primary glomerulonephritis lipid deposits elsewhere. Sudden shut-down of kidneys in response to metabolic insult (infectious disease, toxic agents, drug reactions) or traumatic injury. Clinical symptoms: proteinurea, hematuria, loss of appetite, nausea/ vomiting, fatigue, edema. Replace some of the normal kidney functions through diffusion (waste removal) and ultra ltration ( uid removal) Caused by progressive breakdown of renal tissue, which impairs all renal functions. Clinical symptoms: electrolyte imbalances, nitrogen, retention, anemia, hypertension, weakness, shortness of breath, fatigue, thirst, appetite loss, bleeding, muscular twitching.
