PSYC 2010 Lecture Notes - Lecture 10: Axon Terminal, Fluoxetine, Reuptake

• School: Auburn University
• Department: Psychology
• Course: PSYC 2010
• Professor: Seth A Gitter

Document Summary

Autoreceptors: detect and regulate levels of neurotransmitters in the synapse, receptors on terminal buttons tell sender neurons to stop sending neurotransmitters and it will slow down and eventually stops process of sending neurotransmitters. Enzymes: reabsorption of excess neurotransmitters, sucks back up excess of neurotransmitters (reabsorbs, destroy excess neurotransmitters in the synapse (gap between terminal, chemical binding to excess neurotransmitters making it break down button and dendrite receptors) No longer able to bind to receptor cites. When serotonin is released into the synapse prozac gets absorbed (instead of the serotonin), hence blocking the serotonin from being reabsorbed, and allows serotonin to reach receptor on dendrite of another neuron. 2 major classes: both chemically similar to neurotransmitter, agonist good mimicker . A drug that mimics the effects of a neurotransmitter or blocks its reuptake. Agonist for endorphins (pain regulators: antagonist bad mimicker . Prevents a neurotransmitter"s action in the brain. Like putting wrong key in the wrong lock.