NUR 0020 Study Guide - Final Guide: Glomerular Basement Membrane, Renal Colic, Diabetic Ketoacidosis
Document Summary
Decreases in glucose levels stimulate the production of glucagon which promotes glycogenolysis and gluconeogenesis. Insulin and glucagon are both produced in the pancreas and have most of their effects in the liver. Excess glucose is stored as glycogen in the liver and muscle. Glucose is the only fuel for the brain and nervous system, cns does not store glucose. Decreased levels of glucose stimulate glucagon breakdown of glycogen and breakdown of amino acids and glycerol. Diabetes mellitus: type i (insulin dependent) few or no functional beta cells in the islets of. Develops during childhood (diagnosed btwn ages 5-20) Acute problems with dm appear with infection. Often have symptoms before dx (polyuria, polydipsia- excessive thirst, and polyphagia although lose weight- increased appetite) Genetic factors chromosome 6 (immune system fxn) and 11 (insulin production) Autoimmunity t lymphocytes thought to damage the beta cells (once it starts it takes 3 years to start to see damage)
