NUTR 484- Midterm Exam Guide - Comprehensive Notes for the exam ( 23 pages long!)
Document Summary
Gallbladder: stores and concentrates bile, problems, cholecystitis. Pancreas: produces, h2o2, neutralizes acid, lipases, peptidases, amylases, pancreatitis (inflammation of the pancreas, most is self-correcting if you stop abusing it, biliary stones, biliary sludge, not solid but thick and partially blocks cbd. Idiopathic post-necrotic: cholestasis, hepatitis, fatty liver steatosis, etoh abuse lae(cid:374)(cid:374)ec"s (cid:894)etoh + pem(cid:895, vit a toxicity, non-etoh fatty liver, cirrhosis, symptoms/signs, n/v, anorexia, abdominal distension, epigastric pain, jaundice, ascites, gi bleed esophageal, fector hepaticus ix. Dm: 25. 6 million cases in us, may be 5. 5 million undiagnosed, 7th leading cause of death, associated with many complications, diverse group of disorders that result in increased blood sugar from defect in insulin production, action or both. T1dm: absolute lack of insulin production, beta cell destruction, autoimmune, classic symptoms, polyuria, polydipsia, polyphagia, prone to ketoacidosis. T2dm: 90-95% all cases, more frequent with older age, associated with increased body fat, there is insulin production, but insulin resistance.
