NUR 3145 Lecture Notes - Lecture 6: Agranulocytosis, Euthyroid, Alogliptin
Document Summary
How do we prevent too much?? (negative feedback) This is how we regulate hormone levels. As circulating levels of hormones rise above normal, message is sent to hypothalamus, pituitary gland, or the target organ to shut it down! Produced and secreted by the anterior pituitary gland. Major targets bone and skeletal muscle. Reduces glucose utilization plasma levels rise stimulates insulin release. Most effective for suppressing gh release mimic sst suppression of pituitary. Remember . hypothalamus is the king of homeostasisso hypothalamus senses this imbalance of fluidusually due to increased osmolality in the bloodso hypothalamus then tells the posterior pituitary gland to release adh. Adh acts on the kidneys (renal tubules) to enhance water reabsorption. So more water in the body, osmolality decreases to normal levels, and. Adh also is known as vasopressin because it can cause vasoconstriction at high doses. Neurogenic aka central di: lack of adequate production or secretion of. Adh in the brain treat with desmopressin (ddavp) lifelong.
