NSG 3336 Lecture Notes - Lecture 39: Cerebral Perfusion Pressure, Intracranial Pressure, Cranial Cavity
Document Summary
Pathophysiology = when brain tissue, blood, or csf is increased and there is a loss of intracranial compliance > ability of the body to compensate by adjusting the level of the components. Clinical manifestations > most sensitive indicator is loc. Early signs = progressive confusing and increasing lethargy, sluggish reaction and ovoid shape, focal contralateral motor weakness, and no change in vital signs. Late signs = unresponsive, unilateral or bilateral pupillary dilation with retraction, and contralateral hemiparesis. Cushing"s triad = increase systolic pressure, widening of pulse pressure, bradycardia, and change in respiratory pattern. Intracranial pressure monitoring = can be monitored using a catheter or sensor placed in one of the lateral ventricles of the brain in brain tissue, parenchyma or subarachnoid space. Cerebral perfusion pressure (cpp) = parameter indirectly measure cerebral blood ow and general maintained above 60 mmhg > cpp = map-icp. Ability to monitor pressure and drain csf and considered gold standard due to catheter location tip.
