NS 1150 Lecture Notes - Lecture 16: Low-Density Lipoprotein, Coagulation, Pancreatic Lipase Family

• School: Cornell University
• Department: Nutritional Science
• Course: NS 1150
• Professor: David Levitsky

Document Summary

Accumulation of fat on the walls of our arteries n veins. Bile breaks giant pool of triglycerides into smaller portions. More surface area for pancreatic lipase to work on it. Triglycerides normally too big to fit through membranes. Micell enters intestinal cell for transport to vein. This carries triglyceride into portal vein (blood supply) Transports body very slowly throughout the body until it gets to the liver. Receptors of the 3 types on liver. Searches for receptors on fat cells after it leaves the liver. Lipoprotein lipase on the outside of the fat cell is activated by insulin. Breaks down the fat in vldl into fatty acids. Can be transported into the fat cell. After vldl drops off some of the fat it turns into idl. Can be sent back to the liver and be restocked with more fat. Only has apo-b receptor, not all three.