Nursing 4400A/B Lecture Notes - Lecture 5: Metabolic Acidosis, Hyperkalemia, Ketosis
18 Dec 2017
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Document Summary
Insulin allows our cells to use energy, hormone (storage allows liver to store energy), giving someone glucagon will release that energy. Type 1: no insulin produced, called juvinille diabetes. Type 2: increasing, lack of exercise, western diet high in carbs. Hypo: agitation, anxious, diaphoresis, shaking, hungry, dizzy, coma, pale, confused, blurred vision. Hyper: polyuria, polydipsia, polyphasia, body is trying to flush out the sugars) Dx eval: fasting blood sugars (above 7), random bs (above 11), glucose tolerance test (above 11 after 2 hours), a1c (longer term reflection, over months) Management: focuses a lot on maintaining fuel and insulin, lose weight, and manage diet to avoid insulin, protein gives you staying power. An acute metabolic complication of diabetes occurring when fast are metabolized in the absence of insulin. More likely in t1: there is a complete absence of insulin in t1, therefore more likely to go to ketones (can happen in t2, number of dka in t2 b/c t2 is increasing.
