Kinesiology 2230A/B Lecture Notes - Lecture 3: Cardiac Output, Blood Pressure, Afterload

• School: Western University
• Department: Kinesiology
• Course: Kinesiology 2230A/B
• Professor: Glen Belfry

Document Summary

Hypoxic- reduced vessel diameter, reduced blood flow; without o2. Integrity of contractile proteins is lost, area of heart will not contribute to increase of pressure which leads to ejection of blood. Causes of vascular disease (narrowing of arteries): high bp, smoking, lack of sleep, diabetes, high na, inactivity, stress. Sv increases linearly with exercise but levels off at 40-60% vo2 max in the untrained. Untrained increase initially in sv as intensity increases, between 40-60% vo2 max- sv levels off/might decrease. Trained 40-60%, levels off/may increase when getting to max intensities, sv increases, co increases. Elite sv increases all the way to max, Max hr will be close to the same no matter the training status, rely on increased sv and co. Esv- heart just contracted, during filling volume in ventricle increasing, pressure is very low. Depolarization wave at sa node/av, leading to contraction of ventricles.