Kinesiology 2230A/B Lecture Notes - Lecture 22: Sinoatrial Node, Exercise Intensity, Partial Pressure

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Changes in hematocrit can result from: hemconcentration, blood doping, erythropoietin (epo) With systolic pressure every beat stretches and compresses the walls, over. Systematic arterial pressure is dropping. time is starts to vasodilate and radius starts to increase --> pressure is going to drop. Stroke volume: changes in pressure on arterial side of circulation (vasodilation (increase radius)) -- > stroke volume will decrease --> less blood returning to heart --> you need an increase in hr to maintain same cardiac output: e(cid:454)e(cid:396)(cid:272)ise situatio(cid:374) that is lo(cid:374)ge(cid:396) du(cid:396)atio(cid:374): (cid:272)a(cid:396)dia(cid:272) output (cid:449)o(cid:374)t (cid:272)ha(cid:374)ge. But you have changes on arterial side of circulation (increase radius) causes decrease in pressure --> affects stroke volume (less blood returning to heart) --> need to increase hr to maintain cardiac output. Increased core temperature (start overheating: disrupt enzyme function (optimal activity at around 38 degrees. 40 degrees enzymes will start to denature (bonds within enzymes are broken down) --> enzymes can"t perform and can"t produce atp)

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