BIOD43H3 Lecture Notes - Lecture 21: Exercise Intensity, Evaporative Cooler, Vasodilation

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25 May 2018
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BIOD43 Lecture 21: Biotic and Abiotic Influences on Exercise
Exercise in cold requires higher performance to up metabolis, and produce more heat
Exercise in cold leads to higher VO2 ventilation, this can problematic because it leads to:
-Great heat loss
-Increase sympathetic stimulation= greater glycogen use, and less lipid
-Peripheral vasodilation = lower HR
-Less blood flow to adipocytes
-Decrease muscle enzyme activity
Heat and Exercise
- Evaporative cooling occurs through sweating
ex. Eccrine glands are a sympathetic response and Apocrine glands is sweat produced
from under arm and genital areas, which gets secreted from lipid when bacteria
-Sweating rate is influenced by blood flow
-Nitric Oxid causes local vasodilation
Sweat from athletic is generally less smelly that sweat from nervous people
Exercise in the Heat
- The core temperature at relative exercise intensity is consistent
I ar teperature ou reah aiu ore teperature ith loer asolute
exercise intensities. This limits performae leels
-Increased peripheral vasodilation = TPR and Heart Rate, which is less effective at higher
exercise intensity.
-At highest exercise intensities, vasoconstriction occurs at surface to save blood flow too
active tissue.
Habituation: less of a response (sensation activation) associated with outside influence
-In cold, less shivering to allow better function (ex. Can not sleep well if shivering all the
time)
-In heat, less sweating during early exposure to increase in core temperature
Acclimatization to temp in ectotherms
-membrane viscosity related to temperature
-more membrane viscosity, potentially faster transport, but too viscous is also not good.
Ice fish in polar ice salt water, have better LDH functions when Km value is at -1.9
degrees.
Poiseuille’s La: Q=(P1-P2) pi r4/ 8Ln; CO= HR x SR, the rise in CO is due to rise in HR.
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