PSYC 1000 Lecture Notes - Lecture 5: Chronic Traumatic Encephalopathy, Traumatic Brain Injury, Tau Protein

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Long term consequences are looked at when talking about cte. Traumatic thought to be caused by trauma, such as a mild traumatic brain injury (mtbi), aka concussion. Encephalopathy fancy word for brain pathology: cellular level: terminology. Long, tail-like structure that leads from neuron body to synapses, which connect to the dendrites of downstream neurons. In the cerebral cortex, bundles of axons connecting the bodies of some neurons with the dendrites of others: tbi (traumatic brain injury): cellular level. Short-term: blow to head causes quick stretching of axons (deflated balloon analogy: cte: cellular/molecular level. Long term: affected neurons slowly die and tau proteins are a byproduct: systems level. Memory vs language vs decision making: structure function (refer to table 3. 2 in the textbook) Study of brain damage (human and animal) Functional neuroimaging (where is the brain most active at any given: brain damage time) Accidental (e. g. , stroke) or deliberate (e. g. , neurosurgical lesioning).

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