MICR 2420 Lecture Notes - Lecture 13: Antigen-Presenting Cell, Cell-Mediated Immunity, Cytotoxic T Cell

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Influenza is a virus so you can"t treat it with ciprofloxacin. Exocytosis doesn"t play a role in eliminating an infectious agent and the prms. Pathogen-associated molecular patterns (pamps) are recognized by prms (pathogen recognition molecules) Adaptive immunity begins with antigen presentation to t cells by an antigen presenting cell: ex. Neutrophil macrophage and dendritic cell (in blood neutrophil = the antigen presenting cell, in tissue dendritic: t-helpercell initiates and controls entire rest of the response and keeps stuff in check. T cells have receptors that are antigen specific and b cells have receptors that are antigen specific. One tcell might recognize an epitope and initiate. Once bcell gets activated (starts reproducing) and accumulates huge amounts of er because it needs to secrete antibodies: antibodies: soluble antigen-specific proteins in blood and lymph. Target extracellular structures/pathogens for destruction: complement: non-specific soluble proteins in blood and lymph.

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