NURS 205 Lecture Notes - Lecture 11: Capillary Action, Vascular Permeability, Subcutaneous Tissue
3 Dec 2016
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Document Summary
Wound bed preparation paradigm (dime: first treat cause (e. g. vascular supply, edema, pressure, shear, local wound care, patient-centered concerns (pain, debridement of devitalized tissue & down-loading. Infection (superficial/ deep), inflammation: moisture balance, edge-non-healing wound: biological agents, growth factors, skin substitutes, adjunctive therapies. Healability: healable: underlying cause is corrected, maintenance: lack of sources to support healing, non-healable: cause cannot be corrected. Pressure injuries and health system: as aging pop"n con"t to grow, challenges of preventing and managing pressure ulcers will not abate, pressure ulcers are the most frequent type of expensive, avoidable side-effect of care. Pathophysiology: decreased capillary flow ischemia capillary thrombosis and occlusion of lymphatic vessels. Friction: metabolic demand: pyrexia, sns stimulation ischemia. Complex mechanism: deformation, microclimate, nutritional deficit, ischemia, reperfusion cell death. !variable density !5 layers of squamous cells !migrates/sheds - 14 to 28 days !avascular !ph (5. 0-5. 9): acid mantle !barrier to transepidermal water loss. Iad: incontinence associated dermatitis: diffuse pattern: skin folds, perineum, gluteal form (no pressure)
