MICR 121 Lecture Notes - Lecture 16: Genital Wart, Cervical Cancer, Pap Test

• School: Queen's University
• Department: Microbiology and Immunology
• Course: MICR 121
• Professor: Prameet Sheth

Document Summary

Ti"s (cid:272)a(cid:374) (cid:271)e sp(cid:396)ead from person to person through sexual intercourse (vaginal/anal: oral-genital contact or in other non-sexual ways (kissing*/oral sex, congenital transmission. I. v drugs (sharing needles: wo(cid:373)e(cid:374) te(cid:374)d to (cid:271)e (cid:373)o(cid:396)e (cid:271)iologi(cid:272)all(cid:455) p(cid:396)o(cid:374)e to ti"s tha(cid:374) (cid:373)e(cid:374). Ha(cid:396)(cid:373) (cid:396)edu(cid:272)tio(cid:374) st(cid:396)ategies fo(cid:396) p(cid:396)e(cid:448)e(cid:374)ti(cid:374)g ti a(cid:272)(cid:395)uisitio(cid:374) include: abstinence, practice safe sex (condoms for anal, vaginal, oral sex). Hpv: most common sti in the world. 50% of sexually active individual are infected, majority are asymptomatic. Causes genital warts and cervical cancer (causes almost all cervical ca; second most frequent cancer in women) & incidence: ubiquitous (everywhere); incubation unclear (range: 3 mo several years) Exposure seems to be higher (75% pop"(cid:374) is e(cid:454)posed at some point) Small % develop genital warts; even smaller % develop cancer. Increased risk: sex, age, age of 1st sexual intercourse, co-infections, male circumcision, lifetime # partners. Significant rise in hpv prevalence around age of onset of sexual activity and declines w age.