NURSING 2LA2 Lecture Notes - Lecture 2: Complement System, Platelet-Activating Factor, Innate Immune System
19 Oct 2017
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Document Summary
C-reactive protein degranulation fatty streak granuloma histamine injury. Hdl"s infarct leukotriene migration phagocytosis prostaglandin unstable angina. Chapter 22, disorders of blood flow in the systemic circulation, pp. Inflammation is specific (immune response, memory- acquired) and non-specific (inflammatory response, no memory- innate) Inflammation can be innate (natural/present at birth) or (acquired-evolving over time: each of us will have different immune systems as we are exposed to different pathogens and foreign antigens. Inflammation-2nd line: humoral and cellular responses tell us where the inflammatory response originates, humoral- from blood or plasma. Inflammation: involves complement factors (activated directly by pathogens or indirectly by pathogen-bound antibody, leading to a cascade of), immunity: involves formation and action of antibodies: cells involved: neutrophils, macrophages, lymphocytes. A protective response designed to eliminate the initial cause of injury, remove damaged tissue and generate new tissue. The biochemical and cellular response of vascularized tissue to injury, designed to protect the body from injury.
