MEDRADSC 1B03 Lecture Notes - Lecture 8: Pelvic Inflammatory Disease, Rickettsia, Intracellular Parasite

• School: McMaster University
• Department: Medical Radiation Sciences
• Course: MEDRADSC 1B03
• Professor: sandra charbonneau

Document Summary

Uncoating- external surface removed- viral dna of rna enters host cell nucleus and takes control of host cell dna. Assemble new viruses, move to outer edge and emerge (budding) Release of many new viruses and host cell lysis. Latent stage: some viruses enter host cells, but do not replicate until later. symptoms/signs not present. Mutate: viruses tend to change slightly during replication. Divide by binary fission (like bacteria) but need host cell to replicate. Common std, use host cell to replicate, and causes pid and sterility in females. Elementary bodies: infectious; cell wall, ability to bind. Gets in and becomes rb so it can take over, goes through binary fission or fusion idk which, explode at threshold from all the division, budding (release of eb and rb) Reticulate bodies: noninfectious; use host cell for atp/reproduce. Once large numbers produce rb becomes eb. Pip: pelvic inflammatory disease (can cause adhesion in fallopian tube, stuck together, no egg, sterile)