KINESIOL 3K03 Lecture Notes - Lecture 8: Nociceptor, Hyaline Cartilage, Articular Cartilage Damage
6 Apr 2017
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Document Summary
Contact and non-contact: most acl injuries are non-contact (normally internally applied forces) Amount of muscular control they initially had can impact function (more muscular control is better) Therefore, pain and function is not a direct indicator of what happened. Plant and pivot: rotation, valgus and varus. Multi-plantar stress stop and turn (perpendicular movement?: pain that follows injury: can be extreme unable to walk, and others can, locking knee knee locks into a position (physically stuck in some position) Collateral ligaments (isolated sprain) will have different swelling: different from a 24hr delay which occurs from production of synovial fluid over time filling joint for healing/inflammatory process. A piece of whatever was torn has wedged in between tibia and femur causing. Knee extensor atrophy can occur in 48hrs. Mechanism of injury: valgus movement opening up medial side of knee: happens when there is slight flexion, contact/collision from lateral side of leg, most often (cid:498)contact(cid:499)
