EXSS3024 Lecture Notes - Lecture 2: Resistin, Lean Body Mass, Bariatric Surgery

• School: University of Sydney
• Department: Exercise and Sport Science
• Course: EXSS3024
• Professor: Daniel Hackett

Document Summary

Risk (leads to other conditions), 50-59% of australian(cid:495)s are o/w or obese, cvd, t2d, cancer, insulin resistance (t2d, nafld, metabolic syndrome, polycystic ovary syndrome) What is obesity: bmi classifications, u/w: <18. 5, normal: 18. 5-24. 9, o/w: >25. 0, preobese: 25. 0-29. 9, obese: >30. 0. Iii: >40. 0: weight gain caused by, ageing, tiredness, fluid retention, fat genes, stress, underactive thyroid, steroid treatment, over eating/poor eating, low pa, polycystic ovary syndrome (pcos, diabetes treatment. Pathophysiology/pathogenesis: subcutaneous adiposity (sat), accounts for ~80% body fatness, on outside; keeps us warm & protects us (white, weak correlation b/w sat & disease, correlations, no vldl(cid:495)s, no sbp, no glucose, no insulin, adipose insulin resistance (0. 29) 289 cm2: male, bmi = 24. 8, sat = 172. 2 cm2, vat = 164. 3 cm2, monw (metabolically but overweight obese but normal weight) If have more fat in visceral area but have a healthy bmi = classified as metabolically obese but of normal weight.