BISC 162 Lecture 1: Journal 5 (Bio Lecture)
Document Summary
After vascular damage, hemostasis is the procedure that keeps a closed, high-pressure circulatory system in good repair. Blood extravasation from the circulation and vessel-wall damage quickly start the processes in the blood and the vessel wall that seal the opening. Under typical circumstances, regulatory systems prevent thrombus formation from happening simultaneously with these activities. Excessive amounts of thrombin develop, triggering thrombosis, when pathologic events overcome the hemostasis control mechanisms. In arterial diseases linked to a heart attack and stroke, thrombosis is a crucial occurrence, and venous thromboembolic disorders cause a significant amount of illness and death. Moreover, a thrombus is a blood clot that develops on the inside of the heart or on the walls of blood vessels as a result of the adhesion of blood platelets, proteins, and cells. Blood clots can block veins or arteries, causing thrombosis, which can lead to potentially fatal complications including a heart attack or stroke.
