NURS 3334 Lecture Notes - Lecture 2: Tachypnea, Neonatal Resuscitation Program, Gluconeogenesis
Document Summary
Physiologic transition from intrauterine to extrauterine life. Separation infant from the placenta/first breath events. What left in the lungs after expiration. Negative intrathoracic pressure needed to expand collapsed alveoli (thoracic squeeze. Greatly decreases energy expenditure during breathing during delivery) A lipoprotein (lecithin and sphingomyelin) produced by type ii alveolar cells. Fetal lungs produce sufficient levels surfactant by 34-36 weeks to enable respiration without distress that"s why premature newborns have difficulty breathing on their own. Lung expansion + sufficient surfactant = alveolar stability. When expansion happens and not enough surfactant, everything sticks together. Low surfactant, when baby expands lungs = alveoli clumps together. Allows for pulmonary perfusion required for respiratory exchange. Gasp: response to mechanical, chemical, thermal and sensory changes that allows: Too early baby or neuro disorders = not transition property. Mechanical wall squeezes small amount of fluid from lungs amount of air that replaces fluid this . Chest recoils at delivery creating negative intrathoracic pressure which allows a small.
