NURS 3220 Lecture Notes - Lecture 22: Vagus Nerve, Tachycardia, Low-Density Lipoprotein
Document Summary
Inhibition of raas: sympatholytics, calcium channel blockers, diuretics, other. Circulation: adult volume ~ 5l, 9% pulmonary, 7% cardiac, 84% systemic, left and right side pumps (2), 3 different circulations. If bp remains high, or low, medulla resets to new bp. Constriction of nearly all arterioles (cid:523)leads to (cid:1371) systemic vascular resistance: 2. Constriction of veins (cid:523)(cid:1371) venous return(cid:524: relay information to medulla, if bp drops, vasoconstriction center of medulla: All 3 systems can counter attempts to decrease bp******** Increase stroke volume: so we counter their mechanisms with, 1. Ans- sympathetic input, and baroreceptor reflex countered with: beta blockers. Raas- ace inhibitor, arb, dri, and aldosterone antagonist: renin angiotensin aldosterone. Benign prostatic disease: alpha 2 adrenergic: simulate epinephrine and norepinephrine (sympathetic drugs, beta 2 adrenergic: sympathetic effects of adrenaline on lungs and skeletal muscle, vasodilation of arterioles that supply skeletal muscle, and relaxation of bronchial.
