HSS 3106 Lecture Notes - Lecture 14: Coronary Artery Disease, Calcium Channel Blocker, Angina Pectoris

• School: University of Ottawa
• Department: Health Sciences
• Course: HSS 3106
• Professor: Ron Saulnier

Document Summary

Heart: doesn"t receive oxygen that is it pumping, it needs to pump blood to itself muscular pump with three layers, endocardium - thin inner lining, myocardium heart muscle, epicardium thin outer lining. Development of atherosclerosis fat deposits begin at a young age; it is reversible but the lifestyle is very hard to maintain: calcified plaques are irreversible, can only prevent further accumulation. Ischemia: insufficient amnt of oxygen delivered heart to meet its needs: body effects chest pain, fatigue; like skeletal muscle, cept heart can"t stop pumping. Coronary artery disease: atherosclerosis (hardening of arterial walls due to fatty deposits and plaque) and arteriosclerosis (hardening of arteries regardless of cause) Angina pectoris: episodic pain, reversible oxygen insufficiency; classical (upon physical exertion, goes away with rest), variant, unstable (pain when body is at rest) Types bradycardia < 60 bpm, tachycardia 150-250 bpm, atrial flutter 200-350 bpm, atrial fibrillation >