KINE 1P90 Lecture Notes - Lecture 9: Failure Cause, Low-Density Lipoprotein, Angina Pectoris

• School: Brock University
• Department: Physical Education and Kinesiology
• Course: KINE 1P90
• Professor: David Ditor

Document Summary

The volume of blood ejected by the heart per minute. Hr is regulated through the balance of parasympathetic and sympathetic influence on the sa node. At rest, parasympathetic nerves will release acetylcholline. Ach will bind to receptor on the sa node. Slow down the closure of k+ channels, more k+ will flow out causing hyperpolarization. Since resting potential is farther, it takes longer to reach threshold potential, resulting in slower. Ne binds to receptors on sa node. Speed up rate of depolarization so threshold potential is reached faster causing more frequent action potentials. Speeds up spread of action potential throughout specialized conduction pathways. Volume of blood ejected from ventricles per beat. The more blood that fills the heart, the more that gets ejected by the heart. Frank-starling law the more the ventricles stretch during diastole the more forceful during systole. Control of sv by factors outside the heart (i. e. sympathetic, neural, hormonal)